Occupational exposure to carbon black nanoparticles increases inflammatory vascular disease risk: an implication of an ex vivo biosensor assay

Table 1 Demographics of 106 non-CBPs and 82 CBPs

Variable	Non-CBP	CBP	P value
n	106	82
Age (y, M ± SD)	44.2 ± 5.8	45.7 ± 5.3	0.065^a
Sex (Male, %)	100	100
Race (Han Chinese, %)	100	100
Current smokers (n, %)	69, 65.1	57, 69.5	0.52^b
Packyears (Mdn, Q1-Q3)^d	13.5 (6.0–25)	10 (8–20)	0.34^c
Overweight or obesity (n, %)	52, 49.1	33, 40.2	0.23^b
Urinary OH-PAHs (μg/g cr, Mdn, Q1-Q3)^e	6.34 (2.97–10.92)	4.95 (3.22–7.76)	0.35^c
PM_2.5 (μg/m³, M ± SD, n)	71.0 ± 11.4, 5	800.0 ± 574.5, 16
EC (μg/m³, M ± SD, n)	4.0 ± 0.2, 5	657.0 ± 73.7, 16
Total RNA (μg, Mdn, Q1-Q3)	0.82 (0.47–1.62)	0.75 (0.44–1.69)	0.65^c

CBP Carbon black packer, Q Quartile, M Mean, Mdn Median, PM Particulate matter, EC Elemental carbon, SD Standard deviation
^aStudent t test
^bChi square test
^c Wilcoxon Rank sum test
^dValues in current smokers
^eUrinary OH-PAHs were the sum of 1-hydroxynaphthalene, 2-hydroxynaphthalene, 2-hydroxyfluorene, 2-hydroxyphenanthrene, 9-hydroxyphenanthrene, and 1-hydroxypyrene in urine

ISSN: 1743-8977