Fig. 2From: Amorphous silica nanoparticles accelerated atherosclerotic lesion progression in ApoE−/− mice through endoplasmic reticulum stress-mediated CD36 up-regulation in macrophageExperimental design of SiNPs promoted the atherosclerotic plaque progression in ApoE−/− mice. 4-week-old male ApoE−/− mice fed a Western diet were exposed to SiNPs via intratracheal instillation for 12 times, once per week, in order to investigate the influence of sub-chronic SiNPs exposure on the development of atherosclerosisBack to article page