Fig. 6From: Inhaled ambient-level traffic-derived particulates decrease cardiac vagal influence and baroreflexes and increase arrhythmia in a rat model of metabolic syndromeProposed relationship between cardiac physiology and acute particulate exposure based on susceptibility (e.g., hypertension, MetS, age, heart failure). With increasing concentration and/or exposure time, parasympathetic reflexes may counteract sympathetic dominance, dependent on antioxidant status, pulmonary clearance, vagal integrity, additional gaseous pollutants, and particulate interaction with irritant receptorsBack to article page