Fig. 7From: Diesel Exhaust Particle (DEP)-induced glucose intolerance is driven by an intestinal innate immune response and NLRP3 activation in miceGraphical abstract. Gut exposure to diesel exhaust particles (DEP) shifts intestinal macrophages to an inflammatory state and induces beta-cell dysfunction, which can be restored by anti-IL-1β treatment. DEP: Diesel exhaust particles, PBS: Phosphate-buffered salineBack to article page